18,016 research outputs found

    Strong deflection gravitational lensing by a modified Hayward black hole

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    A modified Hayward black hole is a nonsingular black hole. It is proposed to form when the pressure generated by quantum gravity can stop matter's collapse as the matter reaches Planck density. Strong deflection gravitational lensing happening nearby its event horizon might provide some clues of these quantum effects in its central core. We investigate observables of the strong deflection lensing, including angular separations, brightness differences and time delays between its relativistic images, and estimate their values for the supermassive black hole in the Galactic center. We find that it is possible to distinguish the modified Hayward black hole from a Schwarzschild one, but it demands very high resolution beyond current stage.Comment: 10 pages, 1 figur

    3-Factor-criticality in double domination edge critical graphs

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    A vertex subset SS of a graph GG is a double dominating set of GG if ∣N[v]∩S∣≥2|N[v]\cap S|\geq 2 for each vertex vv of GG, where N[v]N[v] is the set of the vertex vv and vertices adjacent to vv. The double domination number of GG, denoted by γ×2(G)\gamma_{\times 2}(G), is the cardinality of a smallest double dominating set of GG. A graph GG is said to be double domination edge critical if γ×2(G+e)<γ×2(G)\gamma_{\times 2}(G+e)<\gamma_{\times 2}(G) for any edge e∉Ee \notin E. A double domination edge critical graph GG with γ×2(G)=k\gamma_{\times 2}(G)=k is called kk-γ×2(G)\gamma_{\times 2}(G)-critical. A graph GG is rr-factor-critical if G−SG-S has a perfect matching for each set SS of rr vertices in GG. In this paper we show that GG is 3-factor-critical if GG is a 3-connected claw-free 44-γ×2(G)\gamma_{\times 2}(G)-critical graph of odd order with minimum degree at least 4 except a family of graphs.Comment: 14 page

    Opinion formation about childhood immunization and disease spread on networks

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    People are physically and socially connected with each other. Those connections between people represent two, probably overlapping, networks: biological networks, through which physical contacts occur, or social network, through which information diffuse. In my thesis research, I am trying to answer that question in the context of pediatric disease spread on the biological network between households as well as within them and its relationship with information sharing on the social network of households (parents in that case) via Information Cascades. I mainly focus on the Erdos-Renyi network model. In particular, I use two different but overlapping Erdos-Renyi networks for the biological and social networks in the model. I am using agent-based stochastic simulations implemented in MatLab to study the modeling results
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